AQA Syllabus focus:
'Biological explanations of offending behaviour, including genetic explanations.'
Genetic explanations argue that some people inherit a biological vulnerability to offending. Researchers look for patterns of criminal behavior in families and for genes associated with aggression, impulsivity, and antisocial tendencies.
Genetic explanations of offending behavior
Genetic accounts suggest that offending is partly influenced by inherited characteristics. This does not mean there is a single “crime gene” or that genes directly cause criminal acts. Instead, the modern view is that offending is usually polygenic, meaning many genes each make a small contribution to risk.
Genes may affect traits linked to offending, such as impulsivity, aggression, low self-control, or a tendency to seek excitement. These inherited tendencies can increase the likelihood of offending, especially when combined with environmental risk factors such as poor parenting, abuse, or deprivation. Genetic explanations are therefore usually probabilistic, not deterministic: they suggest a greater chance of offending, not certainty.
Evidence from family, twin, and adoption research
One way to test genetic explanations is to see whether offending runs in families. If criminal behavior is more common among biological relatives, this may suggest an inherited influence. However, family studies alone are weak evidence because relatives often share the same environment as well as the same genes.
Twin and adoption studies are more useful because they help separate genetic similarity from environmental similarity.

Standard ACE twin-model diagram illustrating how variation in a trait can be decomposed into additive genetic influences (A), shared environmental influences (C), and non-shared environmental influences (E). It visually encodes the core logic of twin studies: MZ twins correlate 1.0 genetically, DZ twins 0.5, while shared environment is assumed to be 1.0 for both. Source
Twin studies
Twin studies compare monozygotic (MZ) twins, who share 100 percent of their genes, with dizygotic (DZ) twins, who share about 50 percent on average.

Diagram showing how monozygotic (identical) twins arise from one fertilized egg that splits, whereas dizygotic (fraternal) twins arise from two separate fertilized eggs. This supports the key methodological assumption in twin studies: MZ pairs share (approximately) all their genes, while DZ pairs share about half on average. Source
If MZ twins are more alike in offending than DZ twins, this supports a genetic explanation.
Researchers often compare twins using concordance rates.
Concordance rate: the percentage of pairs in which both individuals show the same characteristic, such as a criminal conviction.
A classic study by Christiansen (1977) examined over 3,500 twin pairs in Denmark. He found that the concordance rate for criminal convictions was much higher for MZ twins than for DZ twins, with figures of around 52 percent for MZ twins and about 22 percent for DZ twins. This suggests that genes contribute to offending behavior.
At the same time, the MZ concordance rate was far below 100 percent. This matters because if genes were the only cause, both twins in every MZ pair would offend. The findings therefore imply that environmental influences also matter.
Adoption studies
Adoption studies look at whether adopted children resemble their biological parents or their adoptive parents in criminal behavior. If adoptees are more likely to offend when their biological parent has a criminal record, this supports genetic influence, because they do not share the same home environment.
A well-known study by Mednick et al. (1984) investigated thousands of adopted boys in Denmark. They found that adoptees were more likely to have a criminal conviction if their biological father had one. The risk was even higher when both the biological and adoptive fathers had convictions. This supports the idea that offending may involve both genetic vulnerability and environmental risk.
Adoption research is valuable because it reduces the problem of shared family environment, but it still does not prove that genes act alone.
Candidate genes and molecular genetics
More recent research tries to identify particular genes that may contribute to offending.
Candidate gene: a gene selected for study because researchers think its biological function may be linked to a behavior or disorder.
One widely discussed example is the MAOA gene, sometimes linked with aggressive and antisocial behavior. MAOA affects the activity of enzymes involved in breaking down neurotransmitters. Some variants of this gene have been associated with higher levels of aggression, particularly in people who experienced childhood maltreatment. This suggests that genes may create a vulnerability, but whether that vulnerability is expressed may depend on the environment.

Norm-of-reaction graph demonstrating gene–environment interaction: different genotypes show different outcomes across environmental conditions (non-parallel lines). This is the key idea behind claims that certain genetic variants (e.g., low-activity MAOA) may increase risk mainly under environmental adversity rather than causing offending on their own. Source
Researchers have also argued that offending is not caused by one gene alone. It is more likely to be:
polygenic: many genes contribute small effects
interactive: genes work alongside life experiences
complex: different genes may relate to different forms of offending, such as violent or impulsive crime
This makes simple one-gene explanations too limited for most real criminal behavior.
Evaluation of genetic explanations
Genetic explanations have several strengths.
They are supported by scientific evidence from twin, adoption, and molecular genetic studies.
Findings are often based on objective measures, such as official conviction data.
They explain why offending sometimes appears to cluster in families, even when children are raised apart from biological parents.
However, there are important limitations.
Correlation does not prove causation
Even strong associations between genes and offending do not show that genes directly cause crime. A person may inherit a tendency toward aggression or impulsivity, but whether this becomes offending depends on other influences.
Twin studies may overestimate genetic effects
MZ twins are often treated more similarly than DZ twins, so higher concordance may partly reflect environmental similarity, not just genetic similarity. This is called the equal environments assumption problem.
Adoption studies are not perfect
Adopted children may still experience environments that are not typical, and some are placed with families similar to their biological background. This can blur the distinction between genetic and environmental influence.
Criminal convictions are an incomplete measure
Many studies use official convictions, but not all offenders are caught or convicted. This means researchers may be measuring patterns in detection and sentencing as well as actual offending.
Genetic explanations can be reductionist and deterministic
A purely genetic account risks reducing criminal behavior to biology and ignoring social context, learning, and personal choice. It can also encourage biological determinism, the idea that people are “born criminals.” In reality, the evidence shows that genes usually create risk, not destiny.
Gene-environment interaction is crucial
The strongest modern view is that genes may predispose a person to offend, but environmental stressors often determine whether that predisposition is expressed. This means genetic explanations are useful, but incomplete on their own.
Practice Questions
Identify one finding from twin research that supports a genetic explanation of offending behavior. (2 marks)
1 mark for identifying that monozygotic twins show a higher concordance for offending than dizygotic twins.
1 mark for supporting detail, such as reference to Christiansen or the idea that greater genetic similarity is linked to greater similarity in offending.
Outline and evaluate genetic explanations of offending behavior. (6 marks)
AO1 up to 3 marks:
Genetic explanations argue that offending is partly inherited.
Offending is likely polygenic rather than caused by a single gene.
Relevant evidence may include twin studies, adoption studies, or candidate genes such as MAOA.
AO3 up to 3 marks:
Support from higher concordance in monozygotic than dizygotic twins.
Support from adoption research showing resemblance to biological parents.
Limitation: concordance is less than 100 percent, so environment matters.
Limitation: twin/adoption methods may not fully separate genes and environment.
Limitation: genetic explanations can be reductionist or deterministic.
FAQ
Heritability is an estimate of how much variation in a trait within a population is linked to genetic differences.
Concordance is simpler: it shows how often both members of a pair, such as twins, share a trait.
A high concordance rate does not automatically mean high heritability. Researchers still have to rule out shared environmental influences before making genetic claims.
Epigenetics refers to changes in gene expression without changes to the DNA sequence itself.
This matters because stressful experiences, neglect, or trauma may affect whether certain genes are switched on or off. Two people can share similar genes but show different behavior if their environments influence gene expression differently.
This helps explain why identical twins are not always identical in offending.
Different offenses may involve different underlying traits.
Violent offending is often linked to:
aggression
impulsivity
poor emotional regulation
These traits may show stronger biological components than crimes driven mainly by planning, opportunity, or financial need.
That does not mean violent crime is “genetic.” It means genetic risk may be easier to detect in some offense categories than others.
Candidate-gene studies focus on a small number of genes chosen in advance, but many early findings were difficult to replicate.
Genome-wide studies scan much larger parts of the genome without assuming which genes matter most. This reduces bias and is better suited to complex behaviors influenced by many small genetic effects.
For offending, this approach fits the idea that risk is polygenic rather than based on one or two powerful genes.
It can be discussed, but it is controversial.
Possible uses include:
mitigation during sentencing
support for treatment plans
explaining vulnerability rather than excuse
However, courts are cautious because genetic risk does not prove that a person had no choice. There are also ethical concerns about stigma, privacy, and treating people as dangerous because of their biology rather than their actual behavior.
